Oxidate damage and interaction induced by typical heavy metals in waters on the macrobranchium Nipponense
Abstract
In order to systematically explore the mechanism of chronic biotoxicity of Heavy Metals in waters on a-quatic organics, biological Toxicity bioassayMacrobranchium NipponenseExposed under Cd and Pb were con-ducted in laboratory. superoxide dismutase (SOD), catalase CAT), metallothionein (MT) and malondialdehyde (MDA) were meet used as biomarkers to investigate the single and joint toxicity of Cd and Pb and their interaction on heptapancreas and museums of organics. the results indicated that CD with concentration of 1 mg ยท L-1Can pro-
70.02% when exposed L-1PB for 10 d. most of the CAT activity in heparin can be activated by heavy metal stress. an opportunity tendency of CAT activity change in hepatopancreas and muscle was observed after 10-day exposure. MT and MDA contents in heptapancreas were more sensitive than thought in muscle. moreover, the integrated biomarker response (IBR) reopened that the River Shrimp itself has a certain heavy metal detection function in the time scale through the regulation of enzyme activity, but the oxidative damage cause by heavy metals was not stimulated. the toxicity of single heavy metal was higher than the combined ones. the toxicity of CD to heparin and PB to muscle were the highest. the results obtained could provide the basis for the early warning of ecological risk induced by metals, Development water quality criteria and BaSin Water Environment Man-cement.
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